详细内容（前10条）

1. ⭐ 《自然》子刊：复旦中山医院团队发现，靶向Wnt通路可重编程巨噬细胞，改善肝癌肿瘤微环境，逆转免疫治疗耐药

• ✍️ 作者：奇点肿瘤探秘
• 🏷️ 关键词：肿瘤、免疫、T细胞、巨噬细胞、耐药、通路
• 📝 描述：为靶向BCL9/Wnt克服肝癌免疫治疗耐药提供了全新方向。
• 🔗 查看原文

2. ⭐ 刚刚发表的7.5分生信，针对热点基因集，结合单细胞+机器学习+空转+多重免疫荧光。生信占百分之80实验占20。高效发文章典范！

• ✍️ 作者：生信小课堂
• 🏷️ 关键词：免疫、单细胞、生信
• 📝 描述：点击查看详情
• 🔗 查看原文

3. ⭐ Nature Aging！揭秘逆转免疫衰老新靶点

• ✍️ 作者：芒果师兄
• 🏷️ 关键词：免疫、衰老、aging
• 📝 描述：诱导肿瘤衰老，突破免疫耐药！关键是表观~2025-11-10免疫顶刊！
• 🔗 查看原文

4. ⭐ 《Cell》：癌细胞竟用“痛感神经”远程操控免疫系统！阻断此通路，可实现抗癌与镇痛“双赢”

• ✍️ 作者：生信人
• 🏷️ 关键词：免疫、神经、通路
• 📝 描述：在肿瘤免疫学领域，为什么强大的免疫系统会“失灵”，任由癌细胞生长？
• 🔗 查看原文

5. 深度学习突破跨物种单细胞数据壁垒；FFPE组织样本分析迎来新里程碑；AI与长读长测序联手助力单细胞类型注释 | 单细胞视角

• ✍️ 作者：单细胞天地
• 🏷️ 关键词：测序、单细胞
• 🔗 查看原文

6. 高性价比文章，7.5非肿瘤生信，针对糖酵解的分析并识别关键基因做一下简单的表达验证。发文成本极低！

• ✍️ 作者：生信小课堂
• 🏷️ 关键词：肿瘤、生信
• 📝 描述：点击查看详情
• 🔗 查看原文

7. 组间差异箱线图：单细胞亚群signature在bulk转录组中的应用

• ✍️ 作者：生信技能树
• 🏷️ 关键词：单细胞、转录组
• 📝 描述：单细胞亚群与bulk转录组的联合~
• 🔗 查看原文

8. Cell | 破除争议：不是基因突变！表观遗传信息的丢失是衰老的主要驱动因素

• ✍️ 作者：人体蛋白质组导航计划
• 🏷️ 关键词：衰老、表观遗传
• 🔗 查看原文

9. Cell | 基于大规模脑组织蛋白质组的因果网络解析：阿尔茨海默病关键驱动蛋白图谱

• ✍️ 作者：人体蛋白质组导航计划
• 🏷️ 关键词：蛋白质组、组蛋白
• 🔗 查看原文

10. Nat Aging：刘光慧/王思等发展延缓前列腺衰老的基因疗法

• ✍️ 作者：丁香学术
• 🏷️ 关键词：衰老、aging
• 📝 描述：揭示 GRHL2-CDK19 分子轴驱动前列腺衰老新机制，首创靶向基因疗法成功逆转动物模型衰老表型，为前列腺疾病干预提供全新策略。
• 🔗 查看原文

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详细内容（前10条）

1. ⭐ GSE279475 单细胞 RNA 测序分析揭示了小肠神经内分泌肿瘤的起源细胞以及与早期肿瘤发展相关的基因表达轨迹

• ✍️ 作者：未知作者
• 🏷️ 关键词：tumor、RNA-seq、single-cell、trajectory、regex:intestin(e|al)
• 📝 描述：Contributors : Yoshitatsu Sei ; Joanne Forbes ; Cihan Oguz ; Paul Schaughency ; Gustavo Gutierrez-Cruz ; Faiza Naz ; Stephen R Brooks ; Stefania Dell’Orso ; Pradeep K Dagur ; Jianying Feng ; Xilin Zhao ; Marybeth S Hughes ; Naris Nilubol ; Stephen A WankSeries Type : Expression profiling by high throughput sequencingOrganism : Homo sapiensA hereditary form of small intestinal neuroendocrine tumors (SI-NET) presents with multiple synchronous primary tumors and accompanying tumor precursors at varying stages of development. Capitalizing on this germline property, we conducted single-cell RNA sequencing analysis to identify the cell-of-origin and explore the trajectory of gene expression associated with the early tumor development. The results of the current study may enhance our understanding of the genesis and development of SI-NETs and improve diagnostic, preventive, and therapeutic strategies.
• 🔗 查看原文

2. ⭐ GSE254055 损伤诱导的 CLU 阳性心肌细胞驱动心脏再生中巨噬细胞功能的代谢重编程 [空间转录组学]

• ✍️ 作者：未知作者
• 🏷️ 关键词：macrophage、metabolic、spatial、spatial transcriptomics、transcriptomics
• 📝 描述：Contributors : Lei Fan ; Qi Tang ; Yutong WangSeries Type : Expression profiling by high throughput sequencingOrganism : Mus musculusThe regenerative capacity of the adult mammalian heart is constrained by the post-mitotic nature of cardiomyocytes (CMs). Conversely, neonatal mouse hearts exhibit a regenerative window within the first week of life. Here, we demonstrate that an injury-induced Clusterin-positive (Clu+) CM population reprograms macrophages, facilitating heart regeneration during this timeframe. Clu+ CMs are selectively induced in the border zone of regenerative hearts across multiple species, contrasting with their absence in non-regenerative hearts. Genetic ablation of Clu+ CMs or Clu impedes neonatal heart regeneration, while transplantation of engineered human organoids enriched in Clu+ CMs or Clu overexpression promotes myocardial regeneration in adult mice. Mechanistically, Clu+ CMs secrete CLU, binding to TLR4 in macrophages, directing them toward an immune-suppressive neonatal-like phenotype through Cpt1a-mediated metabolic reprogramming, inducing BMP2 and promoting CM proliferation. Our findings unveil a novel cellular source for mammalian heart regeneration and highlight the fundamental roles of cardio-immune interaction in cardiac repair.
• 🔗 查看原文

3. GSE301847 2-花生四烯酸甘油酯在心血管炎症和心肌重塑中的无偏评估

• ✍️ 作者：未知作者
• 🏷️ 关键词：inflammation、cardiovascular
• 📝 描述：Contributors : Moritz Nöthel ; Susanne V. Schmidt ; Julian Jehle ; Franziska Dorer ; Alexandru Odainic ; Marie Rüthing ; Georg NickenigSeries Type : Expression profiling by high throughput sequencingOrganism : Homo sapiens2-arachidonoylglycerol (2-AG) is an endogenous modulator of inflammatory processes and ligand for the endocannabinoid system. Recent scientific investigations accumulated evidences for its key role in inflammatory cardiovascular diseases, like atherosclerosis and left ventricular remodeling. However, molecular mechanisms set in motion by 2-AG are still poorly understood and its role in cardiovascular inflammation is the matter of controversial debate. Yet, there is a dramatic need to understand local effects of 2-AG at the site of heart inflammation, and tissue injury and reconstruction to develop novel therapeutic strategies which modulate both processes for the better and a fast recovery. Myeloid cells, like tissue-resident macrophages and monocytes are central mediators of inflammatory processes and mediate cardiac adverse remodelling after an insult by myocardial infarction, myocarditis or chronic hypertension. In this study, we investigated the impact of 2-AG on the transcriptional programming of peripheral monocytes as model system for resident macrophages in heart tissue or monocytes which are recruited to the site of cardiac tissue remodelling. Data of RNA sequencing analysis and quantification cytokine expression we seeked to clarify the question, if 2-AG induces rather an inflammatory or a wound healing phenotype of monocytes recruited to the site of cardiac tissue injury.
• 🔗 查看原文

4. GSE307310 簇蛋白-TLR4轴介导的心脏免疫相互作用抑制炎症并触发再生

• ✍️ 作者：未知作者
• 🏷️ 关键词：immune、inflammation
• 📝 描述：Contributors : Lei Fan ; Qi Tang ; Yutong WangSeries Type : Expression profiling by high throughput sequencingOrganism : Mus musculusThis study aimed to investigate the transcriptional effects of BMP2 treatment on neonatal mouse cardiomyocytes. Primary cardiomyocytes were treated with either PBS or BMP2 and subjected to bulk RNA sequencing to profile BMP2-induced changes in gene expression. To further dissect the underlying signaling mechanisms, cells were co-treated with the BMP2 receptor inhibitor Dorsomorphin, allowing assessment of the contribution of the canonical SMAD pathway to BMP2-mediated transcriptional regulation. Comparative transcriptomic analysis across treatment groups provides new insights into the role of the BMP2–SMAD signaling axis in cardiomyocyte gene regulation.
• 🔗 查看原文

5. GSE254058 损伤诱导的CLU阳性心肌细胞驱动巨噬细胞功能代谢重编程，促进心脏再生

• ✍️ 作者：未知作者
• 🏷️ 关键词：macrophage、metabolic
• 📝 描述：Series Type : Expression profiling by high throughput sequencing ; OtherOrganism : Mus musculusThis SuperSeries is composed of the SubSeries listed below.
• 🔗 查看原文

6. GSE254056 损伤诱导的 CLU 阳性心肌细胞驱动心脏再生中巨噬细胞功能的代谢重编程 [立体序列]

• ✍️ 作者：未知作者
• 🏷️ 关键词：macrophage、metabolic
• 📝 描述：Contributors : Lei Fan ; Qi Tang ; Yutong WangSeries Type : OtherOrganism : Mus musculusThe regenerative capacity of the adult mammalian heart is constrained by the post-mitotic nature of cardiomyocytes (CMs). Conversely, neonatal mouse hearts exhibit a regenerative window within the first week of life. Here, we demonstrate that an injury-induced Clusterin-positive (Clu+) CM population reprograms macrophages, facilitating heart regeneration during this timeframe. Clu+ CMs are selectively induced in the border zone of regenerative hearts across multiple species, contrasting with their absence in non-regenerative hearts. Genetic ablation of Clu+ CMs or Clu impedes neonatal heart regeneration, while transplantation of engineered human organoids enriched in Clu+ CMs or Clu overexpression promotes myocardial regeneration in adult mice. Mechanistically, Clu+ CMs secrete CLU, binding to TLR4 in macrophages, directing them toward an immune-suppressive neonatal-like phenotype through Cpt1a-mediated metabolic reprogramming, inducing BMP2 and promoting CM proliferation. Our findings unveil a novel cellular source for mammalian heart regeneration and highlight the fundamental roles of cardio-immune interaction in cardiac repair.
• 🔗 查看原文

7. GSE253859 损伤诱导的 CLU 阳性心肌细胞驱动心脏再生中巨噬细胞功能的代谢重编程 [10X_chromium]

• ✍️ 作者：未知作者
• 🏷️ 关键词：macrophage、metabolic
• 📝 描述：Contributors : Lei Fan ; Qi Tang ; Yutong WangSeries Type : Expression profiling by high throughput sequencingOrganism : Mus musculusThe regenerative capacity of the adult mammalian heart is constrained by the post-mitotic nature of cardiomyocytes (CMs). Conversely, neonatal mouse hearts exhibit a regenerative window within the first week of life. Here, we demonstrate that an injury-induced Clusterin-positive (Clu+) CM population reprograms macrophages, facilitating heart regeneration during this timeframe. Clu+ CMs are selectively induced in the border zone of regenerative hearts across multiple species, contrasting with their absence in non-regenerative hearts. Genetic ablation of Clu+ CMs or Clu impedes neonatal heart regeneration, while transplantation of engineered human organoids enriched in Clu+ CMs or Clu overexpression promotes myocardial regeneration in adult mice. Mechanistically, Clu+ CMs secrete CLU, binding to TLR4 in macrophages, directing them toward an immune-suppressive neonatal-like phenotype through Cpt1a-mediated metabolic reprogramming, inducing BMP2 and promoting CM proliferation. Our findings unveil a novel cellular source for mammalian heart regeneration and highlight the fundamental roles of cardio-immune interaction in cardiac repair.
• 🔗 查看原文

8. GSE306192：用17-β雌二醇或氟维司群处理的MCF-7细胞及其氟维司群耐药衍生细胞的单细胞RNA测序

• ✍️ 作者：未知作者
• 🏷️ 关键词：sequencing、single-cell
• 📝 描述：Contributors : Catherine E Caldon ; Leila EshraghiSeries Type : Expression profiling by high throughput sequencingOrganism : Homo sapiensSingle-cell RNA sequencing (scRNA-seq) on MCF-7 breast cancer cells treated with fulvestrant or 17-β estradiol, and MCF-7 cell line derivatives made resistant to fulvestrant.
• 🔗 查看原文

9. GSE289073 从着床准备到早期妊娠，以单细胞分辨率解码子宫内膜图谱[批量RNA测序]

• ✍️ 作者：未知作者
• 🏷️ 关键词：RNA-seq、single-cell
• 📝 描述：Contributors : Gregory W Burns ; Emmanuel N Paul ; Rong Li ; Manish Persaud ; Kristin Blackledge ; Jessica Garcia de Paredes ; Qingshi Zhao ; Ripla Arora ; Anat Chemerinski ; Nataki DouglasSeries Type : Expression profiling by high throughput sequencingOrganism : Homo sapiensEmbryo implantation relies on the development of a receptive endometrium during the secretory phase of the menstrual cycle. Prior studies have attempted to define the window of embryo implantation within the secretory phase based on patterns of differentially expressed genes, but consensus surrounding this signature has not been achieved. We aim to examine the cell types that characterize the purported receptivity signature and identify new transcriptomic markers that define the mid-secretory endometrium through integrated bulk and single-cell RNA sequencing.
• 🔗 查看原文

10. GSE279106 对 HUVEC 和在有或无 CHIR99021 培养条件下转导 hiHepPC 诱导因子的 HUVEC 进行转录组分析 [RNA-Seq]

• ✍️ 作者：未知作者
• 🏷️ 关键词：RNA-seq、transcriptome
• 📝 描述：Contributors : Shizuka Miura ; Kenichi Horisawa ; Hiroki Inada ; Atsushi SuzukiSeries Type : Expression profiling by high throughput sequencingOrganism : Homo sapiensWe previously showed that a specific combination of three transcription factors (FOXA3, HNF1A, and HNF6) can induce direct reprogramming of human umbilical vein endothelial cells (HUVECs) and peripheral blood-derived endothelial cells into human induced hepatic progenitor cells (hiHepPCs; PMID: 33087715). However, the low induction efficiency of hiHepPCs will be a problem when using them in research and clinical applications. Here, we show that activation of the canonical Wnt signaling pathway increase the reprogramming efficiency of HUVECs to hiHepPCs by rapidly inducing chromatin remodeling and gene expression changes in the transduced HUVECs.
• 🔗 查看原文

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1. 跨癌症亚型靶向 HER2 的进展——泛肿瘤方法

• ✍️ 作者：未知作者
• 🏷️ 关键词：肿瘤、癌症
• 📝 描述：Secret hovertext: HER2（人类表皮生长因子受体 2）是多重实体肿瘤，特别是乳腺癌和胃癌中已确立的治疗靶点。HER2 靶向疗法的开发取得了显著进展，包括单克隆抗体、酪氨酸激酶抑制剂、抗体-药物偶联物（ADCs）以及新型双特异性抗体。这些药物彻底改变了 HER2 阳性转移性癌症的治疗格局，提高了无进展率和整体生存率，并提升了患者的生活质量。除了乳腺癌和胃癌外，HER2 的表达/扩增也被观察到在结直肠肺癌、膀胱癌、卵巢癌和胆道癌等其他实体肿瘤中，并为更大规模患者群体的个性化治疗提供了新机遇。本综述重点介绍 HER2 表达实体肿瘤中 HER2 靶向治疗策略的现状，讨论现有治疗的临床疗效、不良事件特征及挑战。我们探讨新兴治疗方法，包括 HER2 靶向 ADC 等新型药物、联合疗法以及克服耐药机制的策略。此外，我们还探讨了 HER2 表达异质性、生物标志物驱动的患者选择以及诊断工具在患者选择和治疗结果优化中的作用。本综述还探讨了扩展
• 🔗 查看原文

2. 针对巨型细胞增生术进行癌症治疗

• ✍️ 作者：未知作者
• 🏷️ 关键词：癌症
• 📝 描述：Secret hovertext:
• 🔗 查看原文

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1. G4mer——一种用于转录组范围内G-四链体识别的RNA语言模型

• ✍️ 作者：未知作者
• 🏷️ 关键词：transcriptome
• 📝 描述：RNA sequencing reveals how genetic variants alter RNA G-quadruplex formation, using an RNA language model that links structural changes to differences in gene expression…
• 🔗 查看原文

2. 对用于生物学保守单细胞整合的深度学习方法进行基准测试

• ✍️ 作者：未知作者
• 🏷️ 关键词：single-cell
• 📝 描述：Improved deep-learning methods enable more accurate single-cell RNA sequencing data integration by preserving subtle biological variation even within annotated cell types…
• 🔗 查看原文

3. 免疫细胞利用一种令人惊讶的技巧来加速肌肉修复。

• ✍️ 作者：未知作者
• 🏷️ 关键词：immune
• 📝 描述：A research team has found that specific immune cells can connect with muscle fibers in a lightning-fast, neuron-like way to promote healing. These cells deliver quick pulses of calcium, triggering repair within seconds. The mechanism works in both injury and disease models. The discovery could inspire new treatments for muscle recovery and degeneration.
• 🔗 查看原文

4. 一种蛋白质或许是解决白血病治疗失败的关键。

• ✍️ 作者：未知作者
• 🏷️ 关键词：leukemia
• 📝 描述：Scientists have uncovered how leukemia cells manage to escape one of the most commonly used treatments. Over time, these cancer cells subtly change the shape of their mitochondria to avoid dying when the drug tries to kill them. By identifying the protein that controls this shape-shifting, researchers were able to block it in mice, making the treatment powerful again and dramatically extending survival.
• 🔗 查看原文

5. 科学家发现一个可能引发阿尔茨海默病的隐藏弱点。

• ✍️ 作者：未知作者
• 🏷️ 关键词：Alzheimer
• 📝 描述：Scientists have found that a mutation tied to Alzheimer’s disrupts the production and quality of exosomes—tiny cell-made communication packets. Cells with the defective SORLA protein generate fewer exosomes and ones far less able to support nearby brain cells. This weakness may be a key driver of Alzheimer’s development. The research points to new treatment strategies that enhance or restore exosome function.
• 🔗 查看原文

• 柳叶刀子刊发布！北京天坛医院孙涛/张力伟&清华大学廖学斌团队等系统描绘髓母细胞瘤的关键免疫特征，确认B细胞浸润为独立负向预后因素
• 文献分享–人颞叶皮层及白质的单核空间转录组分析揭示与阿尔茨海默病病理的关键关联
• 脑科学 | Nature | 端脑GABA能神经元的转录组学及空间组织
• 综述 | TrendsCancer | 播散性癌细胞休眠的代谢图景
• 一图读懂：主要脂代谢途径
• 国产创新突围！我国团队攻克“可降解心脏封堵器”关键技术
• IF:8.3！南昌大学团队解析细胞互作锁定了GAS6-AXL/MERTK/TYRO3通路！
• 介绍两个生信热点发文思路
• 告别手动调参：广东智慧医学国际研究院开发蛋白质组学全流程自动化解决方案
• 全氨基酸制霸！蛋白测序龙头技术路线图公布！
• 登权威期刊！聚焦食管鳞癌研究：中国医学科学院肿瘤医院团队发现Notch信号促血管生成关键机制
• 虚拟感染能触发免疫反应？《nature neuroscience》揭示大脑的神奇防御机制
• 利用生存预后模型预测癌症患者结局：从数据分析到模型解读
• 顶刊持续发力，探索大脑稳态的奥秘：小胶质细胞与神经元的对话
• STTT ｜ 周光飚教授和于雁教授联手，发布迄今为止最全小细胞肺癌多组学队列数据
• 突破空间多组学整合难题！MultiGATE工具横空出世，精准解码组织微环境与调控网络
• Cell Metabolism: 运动后肌肉恢复的关键密码竟然是它？
• Nat Genet ｜优质代码必学，陆军军医大柴进，北大白凡构建MASLD空间多组学图谱 揭示肝病进展
• 明天下午3点！有奖注册 | 微生物：地球健康的隐形守护者 | Cell Press Live
• 免疫逃逸新机制！武汉大学发文：改善结肠癌抗PD-1疗法的治疗新策略
• 中国药科大学最新Science子刊：开发新型乳腺癌疫苗
• 新晋院士最新Science子刊：揭开多发性骨髓瘤起源细胞，开发CAR-T细胞疗法

• GSE292137 兴奋性神经元 ErbB4 驱动阿尔茨海默病早期病理生理过程
• GSE283032 Satb2 基因在皮层投射神经元中的剂量影响神经元成熟和体感处理 [ChIP-seq]
• GSE301926 核糖体 mRNA 结合通道介导的特化翻译的结构和分子见解 [RNA-Seq]
• GSE275380 离子型AMPA受体通过改变调节性T细胞的分化和功能促进自身免疫性疾病的发生
• GSE272584 单细胞 RNA 测序揭示了 Hdac5fl/fl 和 K14cre;Hdac5fl/fl 小鼠正常皮肤和伤口边缘之间的 RNA 测序基因表达谱，以及细胞异质性和谱系分化轨迹。
• GSE272000 Hdac5fl/fl 和 K14cre;Hdac5fl/fl 小鼠正常皮肤和伤口边缘表皮的 ATAC-seq 分析
• GSE293987 反义寡核苷酸选择性转录组范围评估的工作流程
• GSE288856 克隆 CD8+ T 细胞在软脑膜中定植并与人类退行性脑疾病中的免疫细胞协同作用
• 用于 localis-REX 项目的 GSE265951 RNA-seq
• GSE227543 中性粒细胞分化从早期到晚期的综合多组学和网络分析 [ATAC-Seq]
• GSE227542 中性粒细胞分化从早期到晚期的多组学和网络综合分析[RNA-Seq]
• GSE310460 新发现的增强子对小鼠促卵泡激素β亚基转录的调控[scATAC-seq；小鼠垂体​​]
• GSE310459 新发现的增强子对小鼠卵泡刺激素β亚基转录的调控[ChIP-seq；LβT2细胞]
• GSE310458 新发现的增强子对小鼠卵泡刺激素β亚基转录的调控[ATAC-seq；LβT2细胞]
• GSE305984 小鼠纤维化肺和正常肺支气管肺泡灌洗液刺激人呼吸道基底干细胞的批量RNA测序分析
• GSE305983：TGF-β刺激的Ctrl/PROTAC人呼吸道基底干细胞的批量RNA测序分析
• GSE290822 人类子宫内膜和妊娠早期蜕膜的单细胞图谱揭示了上皮细胞和间质细胞对生育力的不同贡献 [单细胞 RNA 测序]
• GSE282210：年轻和老年小鼠部分肝切除术（PH）后静止肝脏和再生肝脏的转录组分析
• GSE279128 HUVEC 和用 hiHepPC 诱导因子转导的 HUVEC 在有或无 CHIR99021 培养条件下的染色质状态分析 [ATAC-seq]
• GSE239728 ARID1A 招募 TRIM28 进行异染色质的必要重塑，该过程独立于 SWI/SNF [ChIP-seq]
• GSE239727 ARID1A 招募 TRIM28 进行异染色质的必要重塑，该过程独立于 SWI/SNF [RNA-seq]
• GSE311243 AKAP2 协调黏着斑信号支架，促进三阴性乳腺癌的生长和转移
• GSE310807 KLF5 控制胰腺癌中与亚型无关的高相互作用增强子，从而调节细胞存活
• GSE310614 突变型 p53 结合 RNA 导致线粒体功能障碍 [RNA-seq]
• GSE310501 间皮瘤位置影响临床前模型中的肿瘤微环境和免疫检查点治疗反应
• GSE310463 韩国血浆蛋白质组试点研究
• GSE310450 Hippo信号通路调控果蝇表皮色素沉着和多巴胺代谢
• GSE309868 伯克霍尔德氏菌双组分系统 GrtK 和 GrtR 蛋白对细胞内氨代谢解毒的影响。
• GSE300263 靶向转录组方法的直接比较：HTG 与 TempO-Seq
• GSE298083 BRD4抑制通过TXNIP抑制组蛋白H4 UFMylation，从而驱动铁死亡敏感性
• GSE290936 代谢-染色质轴促进人类疟原虫核糖体异质性[II]
• GSE290639 代谢-染色质轴促进人类疟原虫核糖体异质性
• GSE283534 成年小鼠耳蜗和前庭毛细胞的 scRNA-seq 转录组谱 [10_weeks_CBAJ]
• GSE283035 皮层投射神经元中 Satb2 基因剂量影响神经元成熟和体感处理
• GSE283033 Satb2 基因剂量在皮层投射神经元中影响神经元成熟和体感处理 [CUT&RUN]
• GSE282808 MED19 促进 rRNA 的 2’-O-甲基化以促进 IRES 依赖的 c-Myc 翻译
• GSE279380 交感神经元过度激活驱动毛囊坏死和T细胞自身反应性
• GSE277777 高可塑性细胞状态在肺癌中的关键作用